anti mef2b (Santa Cruz Biotechnology)
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Santa Cruz Biotechnology
anti mef2b
Anti Mef2b, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 92/100, based on 15 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+mef2b/MEF-2B+Antibody/pm40640872-74-13-14
Average 92 stars, based on 15 article reviews
Anti Mef2b, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 92/100, based on 15 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+mef2b/MEF-2B+Antibody/pm40640872-74-13-14
Average 92 stars, based on 15 article reviews
anti mef2b - by Bioz Stars,
2026-11
92/100 stars
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other:Article Title: Direct reprogramming of fibroblasts into skeletal muscle progenitor cells by transcription factors enriched in undifferentiated subpopulation of satellite cells Article Snippet: These plasmids were recombined with the CSIV-TRE-RfA-CMV-KT vector by LR reaction (Thermo Fisher Scientific). Article Title: Disruption of chaperone-mediated autophagy-dependent degradation of MEF2A by oxidative stress-induced lysosome destabilization Article Snippet: Anti-MEF2A (sc-313), anti-MEF2B (sc-101097), anti-MEF2C (sc-365862), anti-MEF2D (sc-271153), anti-ACTB (β-actin, sc-47778) and goat anti-mouse (sc-2005) or Article Title: Unique immune cell coactivators specify locus control region function and cell stage Article Snippet: Article Title: Contribution of Myocyte Enhancer Factor 2 Family Transcription Factors to BZLF1 Expression in Epstein-Barr Virus Reactivation from Latency Article Snippet: Anti-MEF2A (TA303858) antibody was obtained from OriGene Technologies, and anti-MEF2B (sc-98594) and Article Title: DNA methylation remodeling in temozolomide resistant recurrent glioblastoma: comparing epigenetic dynamics in vitro and in vivo. Article Snippet: We purchased the following antibodies: antiα-tubulin (Abcam, Cambridge), Article Title: Disruption of chaperone-mediated autophagy-dependent degradation of MEF2A by oxidative stress-induced lysosome destabilization Article Snippet: Antibodies and reagents Anti-MEF2A (sc-313), anti-MEF2B (sc-101097), anti-MEF2C (sc-365862), anti-MEF2D (sc-271153), anti-ACTB (β-actin, sc-47778) and goat anti-mouse (sc-2005) or Article Title: Characterization of a Cardiac-specific Enhancer, Which Directs α-Cardiac Actin Gene Transcription in the Mouse Adult Heart Article Snippet: B, the complexes formed (lane 1) were also probed by preincubation with a preimmune serum (lane 2), with a Article Title: DNA methylation remodeling in temozolomide resistant recurrent glioblastoma: comparing epigenetic dynamics in vitro and in vivo Article Snippet: We purchased the following antibodies: anti-α-tubulin (Abcam, Cambridge), |
![Figure 5. The GME-1 MYC enhancer shows selective transactivation in MYC-intact cell lines and is directly regulated by the OCT2 ternary com- plex. A, Detail of the GME-1 enhancer with ATAC-seq, H3K27 ChIP-seq, and ChIP-seq for OCT2, OCA-B, MEF2B, and p300 in the indicated cell lines. OCT2 BioChIP in BJAB cells is from Hodson and colleagues (39) and represents binding of a transgenically expressed biotinylated OCT2 protein, whereas all other ChIP-seq datasets profile endogenous proteins. Inset at bottom shows further detail of the Phastcons conserved element track for sub-region 195B (see Supplementary Fig. S5E for further detail), with the positions of the conserved OCT2 motif and the genomic duplica- tion identified in the SU-DHL-5 cell line indicated. B, Differential expression of MYC and BCL6 (qRT-PCR) in GME-1–dependent DLBCL cell lines expressing dox-inducible KRAB-dCas9 after transduction with indicated sgRNAs targeting MEF2B, POU2F2, and POU2AF1 (error bars are the SD of five technical replicates per sgRNA; bars are color-coded by measured transcript per legend). C, Transcriptional reporter assays conducted in MYC-intact and MYC::BCL6-LCR+ DLBCL cell lines with conserved subregions of essential MYC and BCL6 NFRs cloned into the STARR-seq_Ori lu- ciferase vector. See Supplementary Fig. S5E–S5H for details of the indicated regions. The signal ratio and statistical comparison for MYC enhancer 195B vs. each BCL6 enhancer is indicated for each cell line (two-tailed Student t test of biological replicates (n = 3); error bars are 95% confidence interval). D, Transcriptional reporter assays in MYC-intact cell lines for constructs bearing the reference NFR 195B enhancer region or the same region with deletion of the OCT2 motif (“195B_MYC_OCT2Del”) indicated in (A) [two-tailed Student t test of biological replicates (n = 3); error bars are 95% CI]. E, Transcriptional reporter assays conducted in HT cells expressing inducible KRAB-dCas9 and transduced with the indicated sgRNAs. Following puromycin selection, cells were electroporated with the indicated constructs and induced with dox for 48 hours prior to Dual-Glo lucifer- ase assay [two-tailed Student t test of biological replicates (n = 3); error bars are 95% CI]. F, Transcriptional reporter assays in MYC-intact cell lines for constructs bearing the reference NFR 195B enhancer region or a clone of the same region from the SU-DHL-5 cell line bearing a partial tandem duplication (“195B_MYC_Dup”) as indicated in (A) [two-tailed Student t test of biological replicates (n = 3); error bars are 95% CI]. **, P < 0.01; ***, P < 0.001; ns, not significant; P ≥ 0.05. FC, fold change; neg ctrl, negative control.](https://doi-unpaywalled-images-cdn.bioz.com/3998/10__1158_slash_2643___3230__bcd___24___0126/10__1158_slash_2643___3230__bcd___24___0126____page9_image1.jpg)